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It is not known if vardenafil is excreted in human sildenafil moa breast milk. Phase 3 clinical trials included more than 834 elderly patients, and no differences in safety or effectiveness of LEVITRA 5, 10, or 20 mg were noted when these elderly patients were compared to younger patients. In volunteers with moderate hepatic impairment, the Cmax and AUC following a 10 mg vardenafil sildenafil moa dose were increased by 130% and 160%, respectively, compared to sildenafil moa healthy control subjects. In volunteers with mild hepatic sildenafil moa impairment (Child-Pugh A), the Cmax and sildenafil moa AUC following a 10 mg vardenafil dose were increased by 22% and 17%, respectively, compared to healthy control subjects. Do not use LEVITRA in patients on renal dialysis as vardenafil has not been evaluated in such patients.
Renal dialysis is not expected to accelerate clearance as vardenafil is highly bound to plasma proteins and not significantly eliminated in the urine.
Consistent with the effects sildenafil moa of PDE5 inhibition on the nitric oxide/cyclic guanosine monophosphate pathway, PDE5 inhibitors, including LEVITRA, may potentiate the hypotensive effects of nitrates.
Do not use LEVITRA in patients who are using a GC stimulator, such as riociguat. PDE5 inhibitors, including LEVITRA may potentiate sildenafil moa the hypotensive effects of GC stimulators.